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A role for complement in glaucoma?

Research output: Chapter in Book/Report/Conference proceedingChapterpeer-review

29 Scopus citations

Abstract

Chronic open angle glaucoma is a degenerative optic neuropathy that can lead to blindness. We have shown that one of the major genes with altered expression in the glaucomatous retina is complement component C1q in both animal models of the disease as well as in humans. These observations together with evidence of upregulation of other complement components within the retina suggest a role for complement in the pathogenesis of this disease. We review the current evidence that supports such a role and discuss possible mechanisms through which complement may act. A thorough understanding of these mechanisms is important in allowing us to rationally design new therapeutic approaches.

Original languageEnglish
Title of host publicationInflammation and Retinal Disease
Subtitle of host publicationComplement Biology and Pathology
EditorsJohn Lambris, Anthony Adamis
Pages95-104
Number of pages10
DOIs
StatePublished - 2010

Publication series

NameAdvances in Experimental Medicine and Biology
Volume703

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