Erratum: ADAMTS9 and ADAMTS20 are differentially affected by loss of B3GLCT in mouse model of Peters plus syndrome (Human Molecular Genetics (2019) 28:24 (4053-4066) DOI: 10.1093/hmg/ddz225)

The authors wish to apologize for errors in B3glct Tm1Nari allele nomenclature in the above article. To be consistent with MGI (J:286130) nomenclature, this corrigendum has corrected nomenclature for B3glcttm1Nari alleles (MGI:727077, MGI:6277078, and MGI:6277079). Materials and Methods Mice and Genotyping The null allele (B3glcttm1.2Nari (MGI:6277079)) is referred to as B3glct-?11–12 (Fig. S2). Supplementary Information Text Supplementary Methods Generation of B3glct mutations in mice. The B3glcttm1Nari allele (MGI:6277077) targeted exons 11 and 12 containing amino acid residues (DDD) essential for catalytic activity of B3GLCT and was generated in C57BL/6 J embryonic stem (ES) cells (1). B3glcttm1Nari targeted ES cells (C57BL/6 J ES cells) were injected into ICR blastocysts to generate chimeras. Refer to Fig. S2 for generation and confirmation of conditional (B3glcttm1.1Nari (B3glct-floxed11–12), MGI: 6277078) and null (B3glcttm1.2Nari (B3glct-?11–12), MGI: 6277079) alleles, and Table S2 for genotyping protocols. (Figure Presented).