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Filifactor alocis Pathogenicity Requires TLR2 and the Oral Microbiome
A. Vashishta
, L. Li
, S. Srivastava
, T. Sharma
, S. Jin
, M. Barati
, D. A. Scott
, R. J. Lamont
,
P. I. Diaz
, S. M. Uriarte
Department of Oral Biology
University at Buffalo
University of Louisville
SUNY Buffalo
Research output
:
Contribution to journal
›
Article
›
peer-review
7
Scopus citations
Overview
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Keyphrases
Pathogenic Potential
100%
Pathogenicity
100%
Filifactor Alocis
100%
Oral Microbiome
100%
TLR2
100%
Inflammation
66%
Microbial Community
66%
Periodontitis
66%
Alveolar Bone Loss
66%
Dysbiotic
66%
Overexpression
33%
Commensal
33%
Gene-deficient Mice
33%
Low Abundance
33%
Inflammatory Markers
33%
Antibody Titer
33%
Germ-free
33%
Alveolar Bone Resorption
33%
Experimental Periodontitis
33%
Osteoclastogenesis
33%
Oral Gavage
33%
Oral Tissues
33%
Oral Microorganisms
33%
Oral Pathogens
33%
Uncharacterized
33%
Oral Microbial Community
33%
Pro-inflammatory Cytokines/chemokines
33%
Immunology and Microbiology
Osteolysis
100%
Pathogenicity
100%
Periodontitis
100%
Alveolar Bone
100%
TLR2
100%
Pathogenic Potential
100%
Oral Microbiome
100%
Inflammation
66%
Infection
33%
Proinflammatory Cytokine
33%
Infectious Agent
33%
Chemokine
33%
Antibody Titer
33%
Germfree Mouse
33%
Infectious Disease
33%
Osteoclastogenesis
33%
Mouth Flora
33%